Breast Cancer Survivors
Sometimes Aspergillus species are opportunistic pathogens causing frank mycosis. These mycoses are collectively called invasive aspergillosis and are usually classified by the portal of entry. Invasive pulmonary aspergillosis is typically manifested as acute pneumonia. Nonpulmonary portals of entry include the eye, paranasal sinus, or gastrointestinal tract or may be associated with vascular surgery and drug addiction. Invasive aspergillosis is largely a disease of a compromised host. Those at high risk include patients with diabetes, drug addicts, chronically ill patients, transplant patients receiving immunosuppressive drugs, and cancer patients receiving chemotherapy. Occupational exposure is also a factor. Historically, the earliest reports of aspergillosis were among wig cleaners and pigeon fanciers. Invasive disease is treated with antifungal chemotherapy, but existing antimycotic drugs have toxic side effects, and the therapeutic outcome is often poor. As the use of...
In an epidemiological survey in Shizuoka-kan (Prefecture) of Japan, a negative correlation exists between tea drinking and stomach cancer. Numerous in vivo studies on mouse or rats were conducted in many countries on the prevention of various cancers with tea extracts or the effective components in tea (such as catechin-type compounds) and their inhibitory effects. Results indicate that tea to a certain degree has preventive and inhibitory effects on stomach cancer, lung cancer, breast cancer, intestinal cancer, liver cancer, and skin cancer.
Induction by Toxins and Therapeutic Agents. Exposure to high levels of toxic chemicals results in necrotic death of the cell. However, long-term exposure at a low level can trigger an apoptotic response. Many of the agents used in chemotherapy exert their affect by inducing apoptosis in tumor and normal cells. Overexpression of genes such as bcl-2 has been linked to resistance to chemotherapy. Clearly, establishing the mechanism of induction of apop-tosis in response to such agents and providing strategies that minimize the affect of antiapoptotic genes should provide novel and more effective chemotherapeutic strategies (42).
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